#Endothelial

The preprint for my main PhD project in the @Vallierlab @SCICambridge is finally available online! And I’m really excited to share it with you all. Follow the tread for some

Rajeev Jayadevan

RajeevJayadevan

Adenovirus-induced thrombocytopenia: the role of von Willebrand factor | Blood | Othman et al American Society of Hematology 2007 |1. Adenoviruses, when given intravenously as in gene therapy, may directly

Critical COVID-19 cases in ARDS show perverse hypoxemia in spite of decent lung compliance.This is odd. Some people have even made far-fetched claims about supposed damage to RBCs or heme,

Chris von Csefalvay

chrisvcsefalvay

The trending wild-ass guess on #COVID19 of the day is this paper by gastroenterologist Farid Jalali, joining a spate of papers that boil down to explaining how respiratory symptoms in

Walter M Chesnut

1) A BIOWEAPON - AN UNPARALLELED DEATH MACHINE: THE SPIKE PROTEIN OF SARS-CoV-2 IS AN ONCOGENIC/NEURODEGENERATIVE VIRAL BIOWEAPON. IT ALSO CAUSES IMMEDIATE ENDOTHELIAL DAMAGE.Make no mistake. The respiratory effects of

1/Why is tricuspid valve (TV) endocarditis more common in those who use intravenous drugs?The portal of entry for most bacteria is the venous system. If quick access to the right-heart

The ScorranoLab

Stephanie Herkenne's paper is finally out! Congratulations to Stephanie, another supertalent I had the privilege to mentor, to all our coauthors on Twitter @MargheZamb @AnnaPellattiero @FonsecaBranco @NatasciaTiso @SorianoMageni @GiacoMarta @Elen

Post-acute "long COVID-19" mostly presents with fatigue, myalgia, shortness of breath, and headaches.The most affected organs are the heart and lungs. Impairment is often worse in formerly hospitalized cases. Neurological

Beta Cuck 4 Lyfe

Covid19 isn't a respiratory virus. That’s what they thought early on because of the pneumonia but it’s a new virus and it took time for them to realize what's up

Constantine Singer

Thread with an even more helpful perspective. This one is entitledWHAT COVID 19 DOES TO SURVIVORShttps://twitter.com/tedcruz/status/1277595193064034312 The estimated death toll for Covid is somewhere between 1 and 2%. Deaths as

DrSimonAshworth

How should we consider #COVID19?This is primarily a vitally induced thrombotic disease spread by droplets Post-mortems tell us the virus causes endothelial damage and overt blood clots in both small

Sharon Moalem, MD, PhD

COVID-19 uses its spike protein as a 'key' to gain entry and unlock a protein called ACE2 to enter human cells. As the ACE2 gene is on the X chromosome,

THREAD from the new #bradykinin research (pre-print) from :"We propose that the severity of the disease and many deaths are due to a local vascular problem due to activation of

Proposed COVID-19 Pathophysiology [Part 3 of (now) 4]: Coagulopathy Strikes Back.What happens when #COVID19 clashes with the

Avraham Z. Cooper, MD

AvrahamCooperMD

1/Why can furosemide improve dyspnea/pulmonary edema from acute congestive heart failure within minutes of administration?The answer does not involve diuresis.#medtwitter #tweetorial 2/Furosemide (aka Lasix) was introduced as a loop diureti

Stage II of Virus and Bacteria Synergistic Infection of Covid-19: A physical model 0. Recap:Virus breaks cell and ECM adhesion, letting the endogenous bacteria in:https://twitter.com/joshux321/status/1264387117200756736?s=20The virus is lar