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#ACE2
Sharon Moalem, MD, PhD
sharonmoalem
COVID-19 uses its spike protein as a 'key' to gain entry and unlock a protein called ACE2 to enter human cells. As the ACE2 gene is on the X chromosome,
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Jaded Court Lady #239842309
Undergroundsar3
Question: The vaccine induced transmembrane spike proteins get expressed and transport to cell surfaces. Can the spikes bind ACE-2R (see center in figure below)? Can vaccine spikes down regulate ACE2
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Dr. Vida Vakilotojar
VidaVakil
If COVID19 virus uses ACE2 receptors on lung cells to invade them, cannot (synthetic) ACE2 be used in an inhaler to protect against the virus? The inhaled ACE2 would deactivate
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Taiwan Formosa Club 🌐
taiwanformosan
Based on #2019nCoV therapy review (https://f1000research.com/articles/9-72/v2), further evidence was found in the 6 genetic variants associated with higher ratio of ACE2 cells. This weaponized coronavirus appears to be optimized for
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Andrew Wong
guavapancake
Truth is a big thing for me, so I'll take some time at the laundromat explaining THE TRUTH.Ordinary people judge if a COVID-19 vaccine is "neutralizing" if you can breathe
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Eliana Golberstein Rubashkyn 👩🔬⚗🧪
ElianaGolber
On January 22 at the beginning of this year I had a suspicion about the protective effect of nicotine on the coronavirus. I have several colleagues in China, I speak
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Ersa Flavinkins
flavinkins
https://pubmed.ncbi.nlm.nih.gov/16166518/ More on ACE2 Tampering by the WIV.https://pubmed.ncbi.nlm.nih.gov/15791205/ https://www.ncbi.nlm.nih.gov/pmc/articles/PMC7111894/ https://pubmed.ncbi.nlm.nih.gov/18448527/ More! The W
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Frieman
MattFrieman
Is there any evidence that the increased levels of ACE2 in either condition enhance the infection of SARS2? Does more ACE2 = more infection in the lung? No evidence this
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Andre Watson 🧬💊💉
nanogenomic
Here is the virus in "closed" (teal) and "open" (green) form. In "open" form, it binds to ACE2. ACE2 also blocks the ideal neutralizing antibody sites, and binds with extremely
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Dr. Angela Rasmussen
angie_rasmussen
New paper shows that smoking cigs increases expression of the #SARSCoV2 receptor ACE2. The idea here is more ACE2, more infection, worse #COVID19.This is a faulty assumption.https://www.atsjournals.org/doi/abs/10.1164/rccm.202003-0693LE#.XqW1P4Cg3FM.
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Nick Jikomes, PhD
trikomes
1/n, Several people asked me about this headline, on a recent preprint claiming that #cannabis extracts high in #CBD have, "the potential of reducing [#coronavirus] infection by 70 to 80
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Sangxa
sangxa
“Smokers may be prone to severe COVID-19 infections, in part, because their lungs contain an abundance of entry points that the virus can exploit.” “COVID-19 infections begin at the ACE-2
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Are you up4BUIA? (Bottom-Up Interdisciplinarity)
Seempleetoo
Results of a bottom-up interdisciplinary approach - COVID19 International analysis checklist - annex: (ACE) polymorphisms hypotheses - v5.0Thank you to all contributors Links of this annex:http://www.ncbi.nlm.nih.gov/pmc/articles/PMC7102561
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Sripal_s
sripal_chandra
The COVID targets- RNA virus. SARS COV2 shares nearly 80% genetic analogy with previous SARS COV particularly for spike glycoprotein. There are two subunits of spike glycoprotein but this is
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Adriaan
A_van_der_graaf
Today we're sharing an analysis of the genetics behind two important COVID-19 genes: ACE2 and TMPRSS2. A thread on what we have found 1/9https://www.medrxiv.org/content/10.1101/2020.04.22.20074963v1 @LifelinesNL @serenasanna1 ACE2 and TMPRSS2 are
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João Rodrigues
jpglmrodrigues
(1/1) On one of our quarantine walks, my girlfriend told me about this tiger in a NY zoo that had tested positive for COVID-19. I was very skeptical and surprised
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