1) SARS-CoV-2 IS A BIOWEAPON THAT HACKS THE HUMAN GENOME. IT DOES THIS BY ACTIVATING ANCIENT VIRAL INSERTIONS WHICH TRANSCRIBE AND RETURN CORRUPTED FILES (GENES) TO OUR DNA - THEY NO LONGER FUNCTION. RETROTRANSPOSONS ARE ITS TOOLS.

Given their capacity to move across the

2) nuclear and mitochondrial genome and recombine, they could mix phenotypes and give rise to infections that may trigger innate and adaptive immune responses by sensing receptors capable of recognising foreign nucleic acids and proteins.

MITOCHONDRIAL DAMAGE

Transposable

3) endogenous retroelements are able to activate the immune system and to epigenetically control the transcription of both nuclear and mitochondrial genes. An example would be Systemic Sclerosis (very similar to Long COVID). In systemic sclerosis, the aberrant transcription of

4) endogenous retroelements shuttling between nucleus and mitochondria could drive oxidative stress, micro-vascular damage, autoimmunity and fibrosis.

RETROTRANSPOSON PATHWAYS DISRUPT MITOCHONDRIAL FUNCTION

Alus belong to a family of retrotransposons or "jumping genes," which

5) copy and paste themselves in new spots in the genome. Alus (exacerbated/initiated by SARS-CoV-2) cause neurodegeneration, compatible with decades of research and offers a plausible mechanism for the disruption of neuronal mitochondrial homeostasis, ultimately cascading into

6) neurodegenerative disease.

IVERMECTIN AND HYDROXYCHLOROQUINE INHIBIT IMPORTIN

These two drugs are efficacious not becuase they attack the virus, but because they stop proteins from being imported EARLY into the cell, and especially, into the nucleus. In other words, they

7) keep the file cabinet of your DNA locked, so the virus cannot get to your "vital documents," edit them and put them back as unintelligible garbage.

THE VIRUS IS AN EXTINCTION LEVEL EVENT

Unless a way is found to prevent the virus from altering DNA, the human species is in

8) certain danger of extinction.

It has been demonstrated the Retrotransposons are active in COVID-19 infection (please see previous Tweet).

https://pubmed.ncbi.nlm.nih.gov/22417684/ 

https://www.sciencedaily.com/releases/2017/03/170308154432.htm

https://www.sciencedirect.com/science/article/abs/pii/S1359610119300784?via%3Dihub https://www.sciencedirect.com/science/article/pii/S1552526017300432