2) “Similar to the proposed mechanism for pulmonary virulence, the pathophysiology for SARS-CoV2 orchitis relies on the primary receptor on Leydig cells in the testis being the ACE2 receptor, facilitating cellular entry”
4) “A mild COVID-19 infection is not likely to affect testis and epididymis function, whereas semen parameters did seem impaired after a moderate infection. SARS-CoV-2 RNA could not be detected in semen of recovered and acute COVID-19–positive men.”
5) “This suggests no viral transmission during sexual contact and assisted reproductive techniques, although further data need to be obtained.”
7) That said semen quality is often temporarily impaired after some infections, so not too abnormal of a finding. Good thing is no inflammation of epididymis. So I don’t think this is that major.
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