2) “Similar to the proposed mechanism for pulmonary virulence, the pathophysiology for SARS-CoV2 orchitis relies on the primary receptor on Leydig cells in the testis being the ACE2 receptor, facilitating cellular entry”
4) “A mild COVID-19 infection is not likely to affect testis and epididymis function, whereas semen parameters did seem impaired after a moderate infection. SARS-CoV-2 RNA could not be detected in semen of recovered and acute COVID-19–positive men.”
5) “This suggests no viral transmission during sexual contact and assisted reproductive techniques, although further data need to be obtained.”
6) Clarification- posts 4-5 refer to study below.

“Eighteen semen samples from recovered men were obtained 8–54 days after absence of symptoms, 14 from control subjects, and 2 from patients with an active COVID-19 infection” https://www.fertstert.org/article/S0015-0282(20)30519-7/fulltext">https://www.fertstert.org/article/S...
7) That said semen quality is often temporarily impaired after some infections, so not too abnormal of a finding. Good thing is no inflammation of epididymis. So I don’t think this is that major.
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