1/

The Cell paper has failed to discuss the most important
IFITM2- Interferon-induced transmembrane protein

These proteins have viral roles, but also
"in Restriction of M. tuberculosis Infection"
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4916766/

And">https://www.ncbi.nlm.nih.gov/pmc/artic... guess what also turns up?

Prevotella... https://twitter.com/sanchak74/status/1251522371200008197">https://twitter.com/sanchak74...

2/

"We found differences in the representation of the genus Prevotella within the Prevotellaceae family, showing significant increase in abundance in IFITM3‐deficient and IFITMdel−/− mice." https://onlinelibrary.wiley.com/doi/full/10.1111/imcb.12000">https://onlinelibrary.wiley.com/doi/full/...

3/
Also, "A host of viruses and pseudoviruses are not restricted by IFITM proteins."
https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4295558/
(not">https://www.ncbi.nlm.nih.gov/pmc/artic... sure if SARS-Cov2 is part of that, pls check)

Here are the proteins sorted on the counts https://sanchakblog.wordpress.com/2020/04/20/highly-expressed-mrna-in-deceased-74-yr-old-covid19-lung-sample/">https://sanchakblog.wordpress.com/2020/04/2...

4/

And ferritin? Everything points to a bacterial infection.

"Gram -ve lung pathogens such as H. influenza have developed strategies to acquire iron by using iron-containing human proteins such as tranferrin, lactoferrin, haemoglobin and ferritin" https://www.ncbi.nlm.nih.gov/pmc/articles/PMC4666040/">https://www.ncbi.nlm.nih.gov/pmc/artic...

And they see low Type I and III interferons and elevated chemokines (true, see above) & thus
"reduced innate antiviral defenses"

but cant make the connection to bacterial infection.