1. This thread describes in detail a novel mechanism of action induced by the MMR vaccine working synergistically with aluminum adjuvant containing vaccines resulting in autoimmunity and brain damage.
2. In the United States children are typically given an MMR vaccine and aluminum adjuvant containing vaccines at the 12 month well-check visit. https://www.cdc.gov/vaccines/schedules/hcp/imz/child-adolescent.html">https://www.cdc.gov/vaccines/...
3. The CDC actually admits here that MMR vaccine does cause brain damage on rare occasions. https://www.cdc.gov/vaccines/hcp/vis/vis-statements/mmr.html">https://www.cdc.gov/vaccines/...
4. I propose that brain damage can occur directly from the innate immune system through a unique interaction induced by the measles component of MMR vaccine administered in combination with aluminum adjuvant containing vaccines.
5. For this interaction to occur, insoluble aluminum particles must cross the blood brain barrier. First, aluminum adjuvant nano particles found in vaccines are phagocytosed (engulfed) by white blood cells called macrophages. https://www.ncbi.nlm.nih.gov/m/pubmed/18496530">https://www.ncbi.nlm.nih.gov/m/pubmed/...
7. Trapped aluminum particles can react with components of the innate immune system throughout the brain. The brain relies heavily on innate immunity and includes virtually all components of complement which are locally produced. https://www.ncbi.nlm.nih.gov/m/pubmed/21546088">https://www.ncbi.nlm.nih.gov/m/pubmed/...
11. These findings support a model in which unwanted synapses are tagged by complement for elimination and suggest that complement-mediated synapse elimination may become aberrantly reactivated in neurodegenerative disease. https://www.ncbi.nlm.nih.gov/m/pubmed/18083105">https://www.ncbi.nlm.nih.gov/m/pubmed/...
13. A membrane attack complex will destroy the cell unless protected by a membrane-bound complement regulator. CD46 acts as a cofactor for C3b and C4b cleavage but it only protects those cells on which it is expressed. https://www.ncbi.nlm.nih.gov/m/pubmed/24161035">https://www.ncbi.nlm.nih.gov/m/pubmed/...
15. The major measles virus receptor CD46 just happens to be the same receptor responsible for regulating complement proteins and preventing MAC formation on host cells that leads to complement-mediated cell lysis (cell death). https://www.ncbi.nlm.nih.gov/m/pubmed/1588280">https://www.ncbi.nlm.nih.gov/m/pubmed/...
18. Due to increased susceptibility, host cells are vulnerable to complement. Interestingly, this is not advantageous for survival of the virus but rather a factor in the attenuation of infection, since it limits the spread of virus. https://www.ncbi.nlm.nih.gov/m/pubmed/8523534">https://www.ncbi.nlm.nih.gov/m/pubmed/...
19. However, over activation of complement triggered by aluminum adjuvant may lead to excessive and uncontrolled activation. Excessive and uncontrolled activation of complement proteins has been implicated in autoimmune diseases. https://www.ncbi.nlm.nih.gov/m/pubmed/28040558">https://www.ncbi.nlm.nih.gov/m/pubmed/...
20. Therefore, aluminum adjuvant combined with vaccine-strain measles virus can induce a synergistic effect on the innate immune system of the brain resulting in damaged tissue directly from complement-mediated cell lysis during excessive and uncontrolled complement activation.
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