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#Hypoxia
Navroop Singh
NavroopSingh_
Combination of Nitrogen Oxide & food rich in Alkaline is critical to boost your body fighting COVID-19. This virus reduces Hemoglobin levels puts the body under depleted O2 levels leading
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The intensivist
Madar_bhagat
My observations1. Tachycardia and thrombocytopenia in OPD is a bad sign. 2. Obese and diabetic can worsen any time in second week even if CT on 6th day has low
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Dan Arendt Pharm.D., BCPS
ArendtDr
I think that acetazolamide and nifedipine may have a role in slowing the progression of #COVID19Yes you read that right.In this #thread we will explore the atypical ARDS that COVID-19
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Mike Coudrey
MichaelCoudrey
NEW RESEARCH: COVID-19 is causing prolonged & progressive hypoxia (starving your body of oxygen) by binding to the heme groups in hemoglobin in your red blood cells.Patients are progressively desaturating
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Casey Bryant, MD
HOGTOWN_DOC
Got an ECMO today consult for refractory and rapidly progressive hypoxia despite intubation. On my arrival, SpO2 was in 30s and 40s. CXR and bedside U/S confirmed total left
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semispinalis
Semispinalis
Every volunteer I know in lko is done. It is impossible to get anything done, impossible to give any help. GOVT DOESN'T CARE. What will beds do? Why should I
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Rod Hayward
ProfHayward
I've been trying to find info on distinguishing death from asphyxia vs. cardiac arrhythmia secondary to hypoxia. Plz forensic med folk weigh. However, IMO this was 2nd degree murder regardless,
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Ed Ostrin, MD, PhD
phlegmMDPhD
There's an emerging theory that the profound hypoxia by COVID is due to a sort of hemoglobinopathy caused by direct viral protein binding to heme. I suppose anything is possible,
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Dr. Rick Pescatore
Rick_Pescatore
Great point. Let’s dissect a few things on deaths of those in restraints.First up: “if you can talk, you can breathe”It takes extremely little respiratory effort to talk—and it’s a
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Scott Gottlieb, MD
ScottGottliebMD
THREAD: There's a growing body of data to strongly suggest #COVID19 predisposes to both venous and arterial thromboembolism due to excessive inflammation, hypoxia, immobilization and diffuse intravascular coagulation. This may
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EMedTox
emedtox
What if this is acute hemoglobinopathy? Reminds me of MetHemoglobinemia-hypoxia out of proportion to symptoms or lung exam or lung imaging. AND 2019-SARS-CoV is active against hemoglobin according to THIS
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Cherie Antoinette
sheriantoinette
Y’all I’ve gotten some great insight regarding COVID. Over the past few nights I’ve made some observations. And COVID is more of a vascular problem than a pulmonary problem, but
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Ari Schulman
AriSchulman
”What’s driving this reassessment is a baffling observation about Covid-19: Many patients have blood oxygen levels so low they should be dead.“Solid overview of the intubation debate:https://www.statnews.com/2020/04/08/doctors-say-ventilators-overuse
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Vineet Chopra
vineet_chopra
There’s a lot of doom and gloom out there when it comes to #CoVID19. It’s true / it’s a bad disease. But we discharged three people home this week after
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Cleavon MD
Cleavon_MD
1918 letter describing Influenza I've treated countless patients with #COVID in NYC and Arizona and I have NEVER seen anything like this deadly virus. 100 years ago during the 1918
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Chandler Marrs
ChanatLucine
I want to talk about 2 papers that highlight the role of mitochondria in what are likely the progression of symptoms assoc with #COVID19. Note these papers are not about
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